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Can Med Assoc J. 1979 August 18; 121(4): 428-30, 433-4. PMCID: PMC1704396

Familial hypogonadotropic hypogonadism with hair loss.

I S Slti and Z Salem

Abstract

In one family several male and female members had hypogonadism and frontoparietal alopecia, whereas other members with normal sexual development had normal scalp hair. Clinical and laboratory evaluation of three affected young men (two brothers and their cousin) revealed that the hypogonadism was the result of decreased serum concentrations of follicle stimulating and luteinizing hormones. There was no evidence of a deficiency of any other pituitary hormone. Long-term treatment of the three patients with human chorionic gonadotropin resulted in an increase in the serum testosterone concentration, the appearance of male secondary sex characteristics and an increase in the size of the external genitalia.
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Eur J Dermatol. 2008 Jul-Aug;18(4):400-6. Epub 2008 Jun 23.

Long-term treatment with finasteride 1 mg decreases the likelihood of developing further visible hair loss in men with androgenetic alopecia (male pattern hair loss).

Kaufman KD, Rotonda J, Shah AK, Meehan AG.

There are no reports on the effects of pharmacologic treatment on the likelihood of developing further visible hair loss in men with androgenetic alopecia (AGA). Our objectives were to examine whether finasteride 1 mg treatment decreases the likelihood of developing further visible hair loss in men with AGA. We conducted an analysis of global photographic assessment data from two Phase III trials in which 1553 men with AGA received finasteride 1 mg/day or placebo for up to 5 years. Finasteride 1 mg treatment led to a 93% decrease relative to placebo in the 5-year likelihood of developing further visible hair loss. We conclude that, in men with AGA, treatment with finasteride 1 mg/day over 5 years led to a marked and sustained decrease in the likelihood of developing further visible hair loss.
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Androgenetic alopecia is caused by heredity. Although it's most common among men, it can also affect women. A history of androgenetic alopecia on either side of your family increases your risk of balding. Heredity also affects the age at which you begin to lose hair and the developmental speed, pattern and extent of your baldness.

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In male hair loss, the hairline recedes as hair thins at the crown. As male pattern baldness progresses, the thinning areas unite, leaving a U-shaped ring of hair around the sides of the head.

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Chemical properties
ATP consists of adenosine and three phosphate groups (triphosphate). The phosphoryl groups, starting with that on AMP, are referred to as the alpha (¥á), beta (¥â), and gamma (¥ã) phosphates. ATP is extremely rich in chemical energy, in particular between the second and third phosphate groups. The net change in energy of the decomposition of ATP into ADP and an inorganic phosphate is -12 kcal / mole in vivo (inside of a living cell) and -7.3 kcal / mole in vitro (in laboratory conditions). This massive release in energy makes the decomposition of ATP extremely exergonic, and hence useful as a means for chemically storing energy.

[edit]
Ionisation of ATP in biological systems
Considering the reaction

HATP3− ¡ê H+ + ATP4−
The acidity constant is Ka = 3.16 x 10−7 giving . In biological systems (cytosol with pH=7.0 or extracellular fluid with pH=7.4), we see that ATP4− is the dominant form (76% of total ATP for pH=7.0).

Space filling image of ATP
3D model of ATP[edit]
ATP metabolism
[edit]
ATP synthesis
ATP can be produced by redox reactions using simple and complex sugars (carbohydrates) or lipids as an energy source. For ATP to be synthesised from complex fuels, they first need to be broken down into their basic components. Carbohydrates are hydrolysed into simple sugars, such as glucose and fructose. Fats (triglycerides) are metabolised to give fatty acids and glycerol.

The whole process of oxidizing glucose to carbon dioxide is known as cellular respiration and is more than 40% efficient at transferring the chemical energy in glucose to the more useful form of ATP.

ATP can be produced by a number of dinstict cellular processes. The three main pathways used to generate energy in eukaryotic organisms are glycolysis, the Kreb's cycle/oxidative phosphorylation, and beta-oxidation.

[edit]
Glycolysis
Main article: glycolysis
In glycolysis, glucose and glycerol are metabolised to pyruvate in the cytosol using the glycolytic pathway. This generates some ATP through substrate phosphorylation catalyzed by two enzymes: PGK and pyruvate kinase. The pyruvate generated by glycolysis can then function as a substrate for the Krebs Cycle.

[edit]
The Krebs Cycle/Oxidative phosphorylation
Main articles: Krebs cycle and oxidative phosphorylation
In the mitochondrion, pyruvate is oxidized by pyruvate dehydrogenase to acetyl-CoA, which is fully oxidized to carbon dioxide by the Krebs cycle. Every turn of the Krebs cycle produces an ATP equivalent (GTP) through substrate phosphorylation catalyzed by Succinyl-CoA synthetase as well as reducing power as NADH.
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C. PLINIUS SEPTICIO SUO S.

(1) Frequenter hortatus es, ut epistulas, si quas paulo curatius scripsissem, colligerem publicaremque. Collegi non servato temporis ordine - neque enim historiam componebam -, sed ut quaeque in manus venerat. (2) Superest ut nec te consilii nec me paeniteat obsequii. Ita enim fiet, ut eas quae adhuc neglectae iacent requiram et si quas addidero non supprimam. Vale.

2

C. PLINIUS ARRIANO SUO S.

(1) Quia tardiorem adventum tuum prospicio, librum quem prioribus epistulis promiseram exhibeo. Hunc rogo ex consuetudine tua et legas et emendes, eo magis quod nihil ante peraeque eodem 'zêlô' scripsisse videor. (2) Temptavi enim imitari Demosthenen semper tuum, Calvum nuper meum, dumtaxat figuris orationis; nam vim tantorum virorum, 'pauci quos aequus ...' assequi possunt. (3) Nec materia ipsa huic - vereor ne improbe dicam - aemulationi repugnavit: erat enim prope tota in contentione dicendi, quod me longae desidiae indormientem excitavit, si modo is sum ego qui excitari possim. (4) Non tamen omnino Marci nostri 'lêkythous' fugimus, quotiens paulum itinere decedere non intempestivis amoenitatibus admonebamur: acres enim esse non tristes volebamus. (5) Nec est quod putes me sub hac exceptione veniam postulare. Nam quo magis intendam limam tuam, confitebor et ipsum me et contubernales ab editione non abhorrere, si modo tu fortasse errori hiar loss treatment nostro album calculum adieceris. (6) Est enim plane aliquid edendum - atque skin care utinam hoc potissimum quod paratum est! Audis desidiae votum - edendum autem ex pluribus causis, maxime quod libelli quos emisimus dicuntur in manibus esse, quamvis iam gratiam novitatis exuerint; nisi tamen auribus nostris bibliopolae blandiuntur. Sed sane blandiantur, dum per hoc mendacium nobis studia nostra commendent. Vale.
7:45 PM

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C. PLINIUS SEPTICIO SUO S.

(1) Frequenter hortatus es, ut epistulas, si quas paulo curatius scripsissem, colligerem publicaremque. Collegi non servato temporis ordine - neque enim historiam componebam -, sed ut quaeque in manus venerat. (2) Superest ut nec te consilii nec me paeniteat obsequii. Ita enim fiet, ut eas quae adhuc neglectae iacent requiram et si quas addidero non supprimam. Vale.

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C. PLINIUS ARRIANO SUO S.

(1) Quia tardiorem adventum tuum prospicio, librum quem prioribus epistulis promiseram exhibeo. Hunc rogo ex consuetudine tua et legas et emendes, eo magis quod nihil ante peraeque eodem 'zêlô' scripsisse videor. (2) Temptavi enim imitari Demosthenen semper tuum, Calvum nuper meum, dumtaxat figuris orationis; nam vim tantorum virorum, 'pauci quos aequus ...' assequi possunt. (3) Nec materia ipsa huic - vereor ne improbe dicam - aemulationi repugnavit: erat enim prope tota in contentione dicendi, quod me longae desidiae indormientem excitavit, si modo is sum ego qui excitari possim. (4) Non tamen omnino Marci nostri 'lêkythous' fugimus, quotiens paulum itinere decedere non intempestivis amoenitatibus admonebamur: acres enim esse non tristes volebamus. (5) Nec est quod putes me sub hac exceptione veniam postulare. Nam quo magis intendam limam tuam, confitebor et ipsum me et contubernales ab editione non abhorrere, si modo tu fortasse errori nostro album calculum adieceris. (6) Est enim plane aliquid edendum - atque utinam hoc potissimum quod paratum est! Audis desidiae votum - edendum autem ex pluribus causis, maxime quod libelli quos emisimus dicuntur in manibus esse, quamvis iam gratiam novitatis exuerint; nisi tamen auribus nostris bibliopolae blandiuntur. Sed sane blandiantur, dum per hoc mendacium nobis studia nostra commendent. Vale.

9:09 PM

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Stroke didn't have any progress for eons.

Press Release Source: AstraZeneca

AstraZeneca Initiates Major Phase III Clinical Trials of CEROVIVE For

Treatment of Patients with Acute Ischemic Stroke

Tuesday May 27, 9:42 am ET

WILMINGTON, Del., May 27 /PRNewswire-FirstCall/ -- AstraZeneca (NYSE: AZN - News) announced today the initiation of two major Phase III pivotal clinical trials to determine the effect of CEROVIVE (R) (also known as NXY-059) on disability and neurological recovery in acute ischemic stroke patients. The clinical trial program, known as the SAINT (Stroke - Acute Ischemic - NXY Treatment) Trials, will compare the efficacy and safety of a 72-hour intravenous infusion of CEROVIVE given within six hours of the onset of symptoms, versus placebo.

Two multinational, multicenter, double-blind, randomized, placebo- controlled parallel-group studies will be enrolling more than 3000 patients. The SAINT-I trial will involve 200 centers across 24 countries in Europe, Asia, Australia and South Africa. The SAINT-II trial will involve patients from approximately 150 sites in the United States, Canada and South America.

"Acute stroke is an area with substantial unmet need for new effective therapies," said Bill Bastain, Global Vice President, Neuroscience Research and Development, AstraZeneca. "AstraZeneca views acute stroke as a treatable emergency and is fully committed to progressing the evaluation of the safety and efficacy of CEROVIVE in large patient populations."

CEROVIVE, a nitrone with free radical trapping properties, is a drug under development that has been licensed from Renovis, Inc. of South San Francisco, California. Initiation of the SAINT trials is based on data from animal models of acute ischemic stroke in both rodents and small primates. In the rodent models, CEROVIVE was administered as late as five hours(1) after ischemia and in the latter models as late as four hours(2) after a permanent ischemic insult, where both short term (3 weeks) and long term (10 weeks) functional outcome was assessed as well as the extent of brain injury. Phase I/II trials including two trials(3)(4) in acute stroke patients have been completed to determine the appropriate dose and tolerability of CEROVIVE. The data from the animal models and phase I/II trials have facilitated the initiation of the phase III trials of CEROVIVE in acute ischemic stroke.

renovis,a registered trademark astrazeneca.

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